Dr. Lamberto Re Explains the Effects of Ozone and NRF2

   

TRULY HEAL

 

Published on Nov 16, 2017

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Dr. Lamberto Re explains the effects of Ozone and NRF2. Ozone triggers NRF2 production which is the key for a cascade of healing reactions. Let's learn more at http://O3academy.com/

Emerson described nrf2 is referred as the master regulator or the antioxidant response moderated desperation of wounder of genes including not only the familiar antioxidant enzymes supersedes disputes catalase.

Nrf2 is a powerful protein that is latent in every cell. It is a switch for 200 or more different genes, all of which code for different aspects of cellular defense such as immune and inflammatory responses, tissue remodeling, fibrosis, carcinogenesis metastasis, and even cognitive dysfunction and addictive behavior.

https://o3academy.com/how-can-ozone-heal-your-body/

Ozone therapy activates Nrf2 pathway zone increases our most potent radical scavenger and cell wall protectors glutathione peroxidase, catalase, and superoxide dismutase.

Ozone therapy disrupts the integrity of the bacterial cell envelope through oxidation of the phospholipids and lipoproteins. In fungi, Ozone inhibits cell growth at certain stages.

With viruses, zone damages the viral capsid and upsets the reproductive cycle by disrupting the virus-to-cell contact with peroxidation zone therapy causes an increase in the red blood cell glycolysis rate.

Ozone therapy leads to an increase in the amount of oxygen released to the tissues. Ozone activates the Krebs cycle by enhancing oxidative carboxylation of pyruvate, stimulating the production of ATP energy.

Ozone therapy increases the production of interferon and the greatest output of tumor necrosis factor and interleukin-2. The production of interleukin-2 launches an entire cascade of subsequent immunological reactions. Activation of the immune system.

Ozone therapy stimulates the neuroendocrine system and therefore produces a feeling of well-being ozone therapy combats aging. Ozone therapy decreases inflammation through the activation of Nrf2 and subsequently the suppression of NFkB pro-inflammatory signaling pathway.

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